OVERCOME YOUR DIABETES TODAY EBOOK
Reverse Diabetes Today Reverse Diabetes Now By Matt Traverso How To Reverse Diabetes Now By Matt Traverso Reverse Diabetes Now How To Reverse. Full text of "Reverse Your Diabetes Today PDF-Book by Matt myavr.info In a test that compared the effect on blood sugar of whole buckwheat groats to. This public document was automatically mirrored from myavr.infoal filename: Reverse Your Diabetes Today PDF-Book by Matt myavr.info
|Language:||English, Spanish, Portuguese|
|ePub File Size:||25.34 MB|
|PDF File Size:||10.52 MB|
|Distribution:||Free* [*Regsitration Required]|
Explains how an ex-diabetic engineer reversed his Type 2 diabetes by following to wellness" today -- and get the Reverse Type 2 Diabetes in 10 Steps ebook. Buy Reverse Diabetes Today - Second Edition: The Scientifically Proven Program to Cure Diabetes and Create Perfect Health (Diabetes Diet - Reversing . Penninghame House The contents of this eBook belong to Penninghame House factors and reverse the disease process. . diet could reverse diabetes.
The hallmarks of insulin resistance syndrome are obesity, particularly central or intra- abdominal obesity , glucose intolerance, or type 2 diabetes mellitus, hypertension , a dyslipidemia characterized by elevated triglycerides , low HDL cholesterol and small dense LDL cholesterol, a hypercoagulable state has been added to the list of characteristic features of the insulin resistance syn- J pected in anyone who has a personal or a Relationship Between Obesity And Type 2 Diabetes Mellitus Last Updated on Fri, 28 Sep Body Weight Animal models of obesity are highly associated with observations of reduced glucose tolerance 61 , and in the non-human primate this glucose intolerance precedes overt type 2 diabetes, as recently Longitudinal in vivo studies in rhesus monkeys have shown that as obese monkeys begin to make the final transition from impaired insulin sensitivity and impaired glucose tolerance at the whole body to overt type 2 diabetes, increasing fasting plasma glucose very closely parallels increasing basal hepatic glucose production In a related event, as hyperinsulinemia progresses to high levels, hepatic extraction of insulin the proportion of the insulin presented to the liver which is removed by the liver declines Nevertheless, at the time of diagnosis of diabetes, insulin levels are usually still elevated above normal, and cell Measurement Of Insulin Resistance Last Updated on Tue, 31 Mar Insulin Resistance Several methods have been used to assess insulin resistance, but most are not readily available or practical for use in clinical practice.
The easiest approach is to measure fasting plasma glucose and insulin concentrations or the glucose and insulin responses during an OGTT or test meal. The higher the insulin concentration in relation to the glucose level, the more insulin resistant the subject.
One can also obtain similar information by measuring the C-peptide concentration in the fasting and stimulated states. Various methods have been developed to analyze glucose and insulin data, one of the most widely used being the homeostasis model HOMA that is most effectively used in large-population studies to assess insulin resistance and beta-cell function 44, A major drawback for using plasma insulin concentrations from individual patients is the wide range of normal values and lack of standardization of insulin assays used by clinical laboratories, both of which make Conclusions fatty acids and insulin sensitivity Last Updated on Wed, 06 Apr Fatty Acids There are plausible biological mechanisms by which excessive provision, cellular uptake and metabolism of fatty acids may lead to impaired peripheral uptake and metabolism of glucose.
These include direct attenuation of glucose-metabolising pathways and or antagonism of the action of insulin at receptor or post-receptor level, or activation inhibition of transcription factors involved in the regulation of glucose and lipid homeostasis at cellular level.
Fatty acid-induced enhancement of glucose-stimulated insulin secretion GSIS appears to be a normal part of the whole body response to excessive fatty acid provision which ensures a compensatory increase in secretion of insulin under such circumstances.
However in the long term this compensatory response can lead to hyper-insulinaemia and, ultimately, failure in beta cell secretion of insulin, leading to diabetes. There is some evidence, from cell and animal studies, that SFA are more likely than other fatty acids to promote an Phosphorylation of a Ser residue near the amino terminus of GSK3 converts that region of the protein to a pseudosubstrate, which folds into the site at which the priming phosphorylated Ser residue normally binds Fig.
This prevents GSK3 from binding the priming site of a real substrate, thereby inactivating the enzyme and tipping the balance in favor of dephosphor-ylation of glycogen synthase by PP1.
Glycogen phosphorylase can also affect the phosphorylation of glycogen synthase active glycogen phosphorylase directly inhibits PP1, preventing it from activating glycogen syn-thase Fig. Although first discovered in its role in glycogen metabolism hence the name glycogen synthase kinase , GSK3 clearly has a much broader role than the regulation of Insulin Changes the Expression of Many Genes Involved in Carbohydrate and Fat Metabolism Last Updated on Wed, 24 Oct Amino Acids In addition to its effects on the activity of existing enzymes, insulin also regulates the expression of as many as genes, including some related to fuel metabolism Fig.
Insulin stimulates the transcription of the genes that encode hexokinases II and IV, PFK-1, pyruvate kinase, and the bifunctional enzyme PFK-2 FBPase-2 all involved in glycolysis and its regulation , several enzymes involved in fatty acid synthesis, and two enzymes that generate the reductant for fatty acid synthesis NADPH via the pentose phosphate pathway glucose 6-phosphate dehydrogenase and 6-phosphogluconate dehydrogenase.
Insulin also slows the expression of the genes for two enzymes of gluconeoge-nesis PEP carboxykinase and glucose 6-phosphatase. These effects take place on a longer time scale minutes to hours than those mediated by covalent alteration of enzymes, but the impact on metabolism can be very significant. When the diet provides an excess of glucose, the resulting rise Insulin resistance and hypertension Last Updated on Tue, 31 Mar Hypertension Hypertension is frequently associated with decreased insulin sensitivity 3 reduced insulin sensitivity has been observed in normotensive offspring of first-degree relatives of hypertensive patients , independent of obesity 4.
Insulin resistance also predates hypertension in normotensive persons. In a prospective investigation of CVD risk factors involving normotensive persons, insulin sensitivity was inversely related to development of hypertension over a 5-yr period 5. This observation has been confirmed in other large studies 6.
There is accumulating data that insulin resistance is associated with abnormalities of the renin-angiotensin system 3,7. For example, the level of insulin resistance in hypertensive persons is influenced by a relatively common polymorphism of the angiotensin-converting enzyme ACE gene, their being a significantly greater insulin resistance with the DD geno type 8.
Recent evidence suggests that tissue overexpression of the RAS leads to Other Risks Of Intensified Insulin Therapy Diabetic Ketoacidosis and Hyperinsulinaemia Last Updated on Wed, 06 Apr Hypoglycaemia Although severe hypoglycaemia was indisputably the major metabolic side-effect of intensive insulin therapy in the DCCT, concerns have been expressed that some intensive treatment regimens may also increase the risk of developing ketosis.
This was primarily related to the use of CSII with insulin pump therapy and was thought to relate to the absence of any intermediate-acting or background insulin in the event of pump failure. In insulin pump therapy, soluble or fast-acting analogue insulin is delivered steadily by a slow infusion of very low doses throughout the day. The insulin delivery is accelerated before meals to deliver boluses, akin to giving intermittent subcutaneous injections of short-acting insulin.
Because the basal insulin is delivered in a very low volume and there is no depot of intermediate-acting insulin in the subcutaneous tissues to act as a reservoir, an interruption in the delivery of insulin can rapidly lead to hyperglycaemia and even ketosis, especially if Heart Disease And Prediabetic States Last Updated on Sun, 14 Oct Insulin Resistance The duration of diabetes influences the development of CAD in patients with type 1 diabetes, but such a relationship has not been demonstrated in those with type 2.
Therefore, it is unclear whether the duration of asymptomatic hyperglyce-mia, or the state of impaired glucose tolerance, may have an important role on the development of CAD preceding the overt manifestations of type 2 diabetes 31, Several studies have shown that the mortality rate due to CAD was higher in patients with impaired glucose tolerance compared to normoglycemic men, although it was smaller when compared to that of patients with overt diabetes.
Просматривать другие видео
At least one study has demonstrated that the risk of CAD increases linearly with fasting blood glucose levels in patients with impaired glucose tolerance, whereas the fasting insulin level has been implicated as a possible independent risk factor for CAD mortality in another study. Early impairment of LV diastolic function has been documented not only in patients Insulin And Oral Hypoglycemic Agents Last Updated on Mon, 17 Sep Body Weight Intensive blood glucose control has, in large intervention trials, been shown to increase body weight in both type 1 and type 2 diabetic patients 82, In the Diabetes Control and Complications Trial DCCT , intensive treatment with either multiple daily injections of insulin or continuous subcutaneous insulin infusion resulted in a 60 increased risk of a body weight more than of the ideal body weight.
On average, the intensively treated patients had a weight gain of 5 kg compared to the patients treated with conventional insulin regimens Most of the weight changes appeared during the first year of treatment 82, In another DCCT substudy it was concluded that the changes in lipid levels and blood pressure that occur with excessive weight gain with intensive therapy were similar to those seen in the insulin resistance syndrome and may increase the risk of coronary artery disease in this subset of subjects with time Not surprisingly, more female than male patients In the aggregate, these risk factors increase the likelihood for sustaining a clinical event and have important prognostic implications.
Endothelial dysfunction, platelet and coagulation abnormalities, and metabolic disorders associated with DM play a major role in accelerating the process of atherosclerosis and generating coronary thrombosis. The interplay of these factors and processes affects healing after arterial wall injury. The diffuse and distal nature of coronary atherosclerosis may contribute to incomplete revascularization and may increase the risk of surgical or percutaneous revascularization in diabetic patients.
Type II diabetics show an inverse relationship between measures of oxidant stress and insulin action , Antioxidant therapy with vitamin E was associated with improved glucose metabolism in some studies of diabetics Collectively, the studies implicate oxidant stress in the metabolic components of insulin resistance observed in subjects with the risk factor cluster.
Insulin Glucose Infusion Last Updated on Wed, 06 Apr Insulin Resistance Long-term mortality in diabetic patients who are hospitalized for acute MI may be reduced by an insulin-glucose infusion followed by multidose insulin treatment. In the Diabetes and Insulin-Glucose Infusion in Acute Myocardial Infarction DIGAMI study, an infusion of insulin and glucose followed by daily subcutaneous injections of insulin resulted in a 52 reduction in mortality within 1 year after myocardial infarction among patients with DM.
This beneficial effect was attributed to improved metabolic control in the presence of an extreme increase in the level of catecholamines in blood and ischemic myocardium that is associated with sudden ischemic episodes. Insulin therapy appeared to beneficially influence acute cardiovascular mortality. A striking reduction in the incidence of fatal reinfarction and left ventricular failure was seen.
These results are consistent with favorable effects in reducing mortality among diverse groups of patients treated with a glucose-insulin -potassium Surgical And Interventional Management Of Cad In Diabetics Last Updated on Fri, 20 Mar Insulin Resistance Several clinical trials have demonstrated that outcomes after myocardial revas-cularization are different in diabetic patients compared to nondiabetic patients.
These differences should influence treatment decisions. Over the last decade, percutaneous coronary intervention PCI has gained increasing acceptance as an alternative to CABG surgery in selected patients. However, several reports demonstrating reduced long-term survival in diabetic patients treated with standard coronary balloon angioplasty have led to concerns regarding the use of PCI in this group of patients.
A complete understanding of the mechanisms responsible for the reduced survival is of critical importance in the management of diabetic patients.
How To Reverse Your Diabetes Today!-Live A Better Life
The operative and procedural management of diabetics with symptomatic CAD will be discussed based on the type of intervention employed. Diabetic women in particular have a relatively poor prognosis, in part related to an increased incidence of congestive heart failure, rein-farction, and recurrent ischemic events Late mortality is significantly increased in diabetic compared with nondiabetic patients Stress hyperglycemia at the time of MI is strongly correlated with mortality rates.
In a meta-analysis conducted by Capes et al. Results from retrospective studies and registries bear on the role of CABG in diabetic subjects.
A caveat in the interpretation of these results is that such databases of diabetic patients who have undergone coronary intervention may not be generalizable to more unselected groups. Further, the prognosis in such nonrandomized cohorts can be influenced The disease was known to be connected somehow with the pancreas, a gland located just below the stomach see Figure Today we know that diabetes mellitus is caused by a lack of the protein hormone insulin type I or juvenile onset diabetes or by a lack of insulin receptors on the target tissues type II or adult onset diabetes.
For patients in which the hormone is lacking, insulin replacement therapy is an extremely successful treatment. At present, more than 1. Mellitus Greek for honey reflects the fact that the urine of an untreated diabetic is sweet.
Insulin binds to a receptor on the plasma membrane of a Thus, there are relatively few patients with CHF in whom the only predisposing factor is diabetes.
He outlines the reasons for T2 and tells what he did to "cure" his diabetes thereby passing on his methods to the reader through his "Wellness" program. His background in biochemistry and medicine when relating to diabetes is expert.
I highly recommend this book to those who wish to be in super control and possibly rid themselves of this dread disease. After trying the program for just 3 days, my BS levels dropped points and are still going down. I am hopeful that I will have a full reversal and will someday be medication free.
This is a simple program to follow, that will change your life completely. Your body will be fed the foods it needs to heal itself and you will no longer crave the "dead" foods that Mr. McCully speaks of in his book.
I can't thank Mr. McCulley enough for writing this book and sharing his own near death experience with diabetes. It's just amazing to see my BS levels dropping daily. When diagnosed in Oct , my BS was and with meds and the doctors direction, I couldnt get it below on most days. I took it this morning, after being on Mr. McCulley's program now for 10 days and my BS level was I think that's pretty incredible.
I feel as if I have control over my sugar for the first time since being diagnosed and I owe it all to Mr. William Bayer Rochester, NY I recommend this book not only to all diabetics but to anyone who is suffering from his or her unhealthy lifestyle. Although Mr. McCulley's theories are not strictly in line with current medical thinking, his advice for changing our lives for the better is right on.
Over half of all Type 2 diabetics who strictly follow his advice will be cured of their diabetes problem, as well as lowering their cholesterol and blood pressure. Don't miss out on this book. What do doctors have to say about Death to Diabetes? Some of them are reducing their medication and others are coming off their medication. There's something about those engineers. I plan to keep this book handy in my office.
Buy the book! I think that it is outstanding. What you wrote is not taught in medical school, that's why some physicians may not support it.
Don't worry my friend, the Public will support you, because you have done a superb job of researching, treating yourself and putting it in writing.
Let the book speak for itself and you. Whenever I see a diabetic, I recommend your book to them. Those that WATCH Things Happen as their bodies rot from the inside, leading to amputation, blindness, kidney failure, heart attack and stroke How did the author discover how to beat and reverse Type 2 diabetes?
DeWayne McCulley is an ex-diabetic engineer who survived a near-death, diabetic coma with a blood glucose level of -- more than points above normal!
Despite two blood clots, high cholesterol, pneumonia and four insulin shots a day, DeWayne was able to use his engineering and biochemistry background to wean himself off the insulin, Coumadin and Lipitor -- to reverse his Type 2 diabetes. He lowered his average glucose level to With a lot of encouragement from his daughter, his mother and people from work, the local churches, wellness industry, and the ADA diabetic support group he was facilitating, DeWayne decided to write the book, Death to Diabetes.
This book explains his experience with diabetes, the real root causes of diabetes, and the pathogenesis of the disease. His hope is that this book and his wellness training programs will inspire you the way he was inspired -- by people he never would have met if it weren't for his experience with diabetes. Are you ready to start your journey from illness to wellness? If diet and exercise are supposed to be the keys to controlling diabetes, why are so many people still struggling with their diabetes?
There are 7 reasons: Most people including doctors don't really understand diabetes. As a result, if you don't understand the problem, you can't solve the problem.
There is so much myth and misinformation, that most diabetics think they understand. So, when their health doesn't improve, they blame themselves or their doctors.
What is the main cause of type 2 diabetes?
We deny that we have diabetes, leading to no action as the disease takes hold in our bodies. We believe that the drugs are working because our blood sugar is lower and we don't feel any discomfort. We believe that our doctor and his drugs are going to save us from the amputation, blindness, kidney failure, heart attack, and stroke.
We don't like changing our lifestyle, especially when it comes to eating and exercise. We have doubt that anyone could do something that a doctor couldn't do.
But, if we did the in-depth research, the doubt would go away very quickly. The ebook Reverse Type 2 Diabetes in 10 Steps addresses all of these areas -- because it was written by an ex-diabetic who struggled with the exact same issues that you and other diabetics are struggling with right now! What is Type 2 Diabetes If your goal is to get your Type 2 diabetes under control, beat your diabetes, reverse your diabetes, or possibly cure your Type 2 diabetes, then is critical that you acquire the proper knowledge to understand how this disease works.
Type 2 diabetes non-insulin-dependent diabetes mellitus, NIDDM is a metabolic disorder where the cells of the body have lost the ability to effectively utilize the insulin produced by the pancreas. This is known as insulin resistance. Type 2 diabetes used to be called adult-onset diabetes, because it primarily affected older adults. But today with more children being overweight and sedentary, Type 2 diabetes is affecting them as well as adults.
Type 2 diabetes is a combination of insulin resistance, inflammation, toxicity, cellular dehy-dration, nutrient starvation, and oxidative stress that affects trillions of cells.
In other words, type 2 diabetes is more than just a "blood sugar" disease!
Fix the cells, you fix the disease! This is important to understand because once you must realize that you need to "fix the cells" and do more than just "control your blood sugar" , then, you're on the way to understanding how to beat and reverse your diabetes.
You also realize that the drugs which only try to control your blood sugar won't help you to beat the disease. Insulin resistance and inflammation prevent your body's cells from effectively using the insulin produced by the pancreas. That is, the insulin receptors on the surface of each cell are damaged inflamed , ignoring the presence of insulin in your blood and refusing to allow glucose from your blood to enter your cells.
The cells in your body require the glucose as fuel in order to produce energy.
Without this fuel, your cells cannot produce energy and perform their functions. Over a period of years, the insulin resistance and inflammation spreads to more and more cells, leading to an increase in the production of fat cells and the need for more insulin from those cells. This increases the fat storage, especially in the abdomen area, while inhibiting fat metabolism because of the excess insulin known as hyperinsulinemia.
Over a period of years, this may lead to beta cell dysfunction, which may reduce insulin production and cause blood glucose levels to rise even further. In addition, there is an increase in oxidative stress, glycation, and a depletion of key micronutrients such as potassium, chromium, Vitamin C, and Vitamin E causing a severe nutrient deficiency.
All of this dysfunction leads to more cell membrane damage and a buildup of homocysteine, which causes damage to the artery walls. This leads to arterial plaque formation, an increase in blood viscosity and blood pressure, and, in some cases, an increase in cholesterol.
Type 2 Diabetes Cure: 4 Ways to Reverse the Disease
The good news is that these biological changes can be corrected with the proper nutrition, exercise, and spiritual health. If you are overweight, a Type 2 Diabetic, or concerned about becoming diabetic, this book will provide you the knowledge for optimum diabetes-management, enabling you need to combat and defeat this disease.Either way, it's a wonderfully alkaline soup, packed with chlorophyll.
Assemble fiesta tacos with layers of refriend beans, salsa, guacamole, red peppers and mixed greens. Besides providing that fat, the avocado is the key to the creaminess of this smoothie. Axe on Twitter Dr. Do not use center stem. Simmer 10 minutes.